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Joined: Jun 2008
Yes, balls have recently emerged.
I went to pick him up the other day when I realised I could feel balls on my hand, he looked up at me as if to say "You do realise you are touching my balls?"
We need to punish the French, ignore the Germans and forgive the Russians - Condoleezza Rice.
The Antagonist
Unregistered
OK to answer your coat color question:
From Wikipedia:
http://en.wikipedia.org/wiki/Tortoiseshell_cat
Quote:Main article: Cat coat genetics
Tortoiseshell and calico coats result from an interaction between genetic and developmental factors. The primary gene for coat color © is located on the X Chromosome and has two co-dominant alleles, Orange (XO) and Black (XB), that produce orange phaeomelanin and black eumelanin pigments, respectively. The cells of female cats, which like other mammalian females have two X Chromosome (XX), undergo the phenomenon of X-inactivation, in which one or the other of the X-chromosomes is turned off at random in each cell in very early development. The inactivated X becomes a Barr body. Cells in which the chromosome carrying the Orange (XO) allele is inactivated express the alternative Black (XB) allele. Cells in which the Black (XB) allele is inactivated express the Orange (XO) allele. Pigment genes are expressed in melanocytes that migrate to the skin surface later in development. In bi-colored tortoiseshell cats, the melanocytes arrive relatively early, and the two cell types become intermingled, producing the characteristic brindled appearance consisting of an intimate mixture of orange and black cells, with occasional small diffuse spots of orange and black.
A famous calico, TamaIn tri-colored calico cats, a second gene interacts developmentally with the coat color gene. This spotting gene produces white, unpigmented patches by delaying the migration of the melanocytes to the skin surface. There are a number of alleles of this gene that produce greater or lesser delays. The amount of white is artificially divided into mitted, bicolor, harlequin, and van, going from almost no white to almost completely white. In the extreme case, no melanocytes make it to the skin and the cat is entirely white (but not an albino). In intermediate cases, melanocyte migration is slowed, so that the pigment cells arrive late in development and have less time to intermingle. Observation of tri-color cats will show that, with a little white color, the orange and black patches become more defined, and with still more white, the patches become completely distinct. Each patch represents a clone of cells derived from one original cell in the early embryo.
Male cats, like other mammalian males, have only a single X chromosome (XY) that does not undergo X-inactivation: coat color is determined by which allele is present on the X, and they will be either entirely black or orange. Very rarely (approximately 1 in 3,000[3]) a male tortoiseshell or calico is born. These animals typically have an extra X chromosome (XXY), a condition known in humans as Klinefelter syndrome, and undergo an inactivation process like that in females. As in humans, these cats are almost always sterile because of the imbalance in sex chromosomes. Some male calico or tortoiseshell cats may be chimeras, which result from the fusion in early development of two embryos with different color genotypes. Others are mosaics, in which the XXY condition arises after conception and the cat is a mixture of cells with different numbers of X chromosomes.
And from another site: (lots of photos)
http://www.seregiontica.org/Colors/torti...torbie.htm
